血液檢測沙眼衣原體IgG、IgM抗體診斷試劑

血液檢測沙眼衣原體IgG、IgM抗體診斷試劑

廣州健侖生物科技有限公司

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血液檢測沙眼衣原體檢測卡

血液檢測沙眼衣原體檢測卡

血液檢測沙眼衣原體IgG、IgM抗體診斷試劑

二維碼掃一掃

【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
【騰訊 】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-3室
【企業文化】

晚期因肝硬化引起門靜脈高壓和長期淤血，致脾臟呈進行性腫大，有的病人腫大的脾臟可占據大部分腹腔甚至下抵盆腔，并伴有脾功能亢進現象。鏡檢可見脾竇擴張充血，脾髓內、血管周圍及脾小梁的結締組織增生，脾小體萎縮減少，中央動脈管壁增厚發生玻璃樣變。脾臟中偶有蟲卵發現。4．其它臟器病 在胃及腸系膜以及淋巴結、胰、膽等偶有蟲卵沉積。血吸蟲病株儒患者有腦垂體前葉萎縮性病變和壞死，并可繼發腎上腺、性腺等萎縮變化，骨骼發育遲緩，男子有睪丸退化，女子有盆腔發育不全。異位性損害主要由于急性感染時大量蟲卵由靜脈系統進入動脈，以肺和腦的異位損害為多見。肺部可有大量蟲卵沉積和發生出血性肺炎。腦部病變多見于頂葉皮層部位，腦組織有肉芽腫和水腫。臨床表現編輯侵襲期自尾蚴侵入體內至其成熟產卵的一段時期，平均1個月左右。癥狀主要由幼蟲機械性損害及其代謝產物所引起。在接觸疫水后數小時至2～3天內，尾蚴侵入處有皮炎出現，局部有紅色小丘疹，奇癢，數日內即自行消退。當尾蚴行經肺部時，亦可造成局部小血管出血和炎癥，患者可有咳嗽、胸痛、偶見痰中帶血絲等。另外未抵達門脈的幼蟲被殺死后成為異體蛋白，引起異體蛋白反應，而出現抵熱、蕁麻疹、嗜酸性粒細胞增多等表現。急性期本期一般見于初次大量感染1個月以后，相當于蟲體成熟并大量產卵時期。大量蟲卵沉積于腸壁和肝臟；同時由于蟲卵毒素和組織破壞時產生的代謝產物，引起機體的過敏與中毒反應。臨床上常有如下特點：1．發熱：為本期主要的癥狀，發熱的高低，期限和熱型視感染輕重而異。熱型不規則，可呈間歇或弛張熱，熱度多在39～40℃，同時伴有畏寒和盜汗。發熱可持續數周至數月，輕癥患者的發熱較低，一般不超過38℃，僅持續數日后自動退熱。2．胃腸道癥狀：蟲卵在腸首，特別是降結腸，乙狀結腸和直腸大量沉積，造成急性炎癥，患者出現腹痛和腹瀉。由于腸道嗜酸性膿腫，可引起表層粘膜壞死形成潰瘍，故常呈痢疾樣大便，可帶血和粘液。
Late due to cirrhosis caused by portal hypertension and long-term congestion, resulting in progressive splenomegaly, and some patients with enlarged spleen can occupy most of the abdominal cavity or even down to the pelvic cavity, accompanied by hypersplenism. Microscopic examination revealed splenic sinus dilatation and hyperemia, connective tissue hyperplasia within the spleen, intraperitoneal and splenic trabecular, atrophy of splenic corpus, and thickening of the central arterial wall with hyalinization. Occasionally, eggs are found in the spleen. 4. Other organ diseases Occasionally eggs are deposited in the stomach and mesentery as well as in lymph nodes, pancreas, and gall bladder. Schistosomiasis patients with atrophy have anterior pituitary atrophy and necrosis, and can be secondary to adrenal gland, gonadal and other atrophy changes, skeletal growth retardation, men with testicular degeneration, women with pelvic hypoplasia. Allosteric damage is mainly due to a large number of eggs from the venous system into the arteries during acute infection, and ectopic lesions in the lungs and brain are more common. The lungs can have a large number of eggs deposited and hemorrhagic pneumonia. Brain lesions are more common in the parietal cortex, with granulomas and edema in brain tissue. The clinical manifestation of the editorial invasion period is about 1 month from the time when the tail lice invade the body to mature mating. Symptoms are mainly caused by larval mechanical damage and its metabolites. In a few hours to two to three days after contact with the water, there was dermatitis at the site of the infestation of the cercariae, and there were red small papules and itchiness in the area, which spontaneously subsided within a few days. When the cercaria passes through the lungs, it can also cause localized small blood vessel hemorrhage and inflammation. The patient may have cough, chest pain, and occasionally sputum blood stains. In addition, the larvae that have not reached the portal vein are killed and become allogeneic protein, causing allogeneic protein reactions, and there is resistance to heat, urticaria, and eosinophilia. The acute phase of this period is generally seen in the first large-scale infection after 1 month, equivalent to the maturation of the body and a large number of spawning period. A large number of eggs are deposited in the intestinal wall and liver; at the same time, the body's allergic and poisoning reactions are caused by the toxins and metabolites produced by tissue destruction. Clinically, there are often the following characteristics: 1. Fever: The main symptoms of this period, the level of fever, duration and heat depending on the severity of infection. Irregular heat, can be intermittent or remittent fever, heat at 39 ~ 40 °C, accompanied by chills and night sweats. The fever lasts from several weeks to several months. Mildly ill patients have low fever. Generally, they do not exceed 38°C. They only automatically heat down after a few days. 2. Gastrointestinal Symptoms: Eggs are deposited in the intestine, especially in the descending colon, sigmoid colon, and rectum, resulting in acute inflammation, with abdominal pain and diarrhea. Due to the eosinophilic abscess in the intestine, it can cause ulceration in the surface mucosal necrosis, so it often presents with diarrhea-like stools, which can bring blood and mucus.